Download Alzheimer's disease and related disorders annual, 2002 by Jeffrey L Cummings, Serge Gauthier PDF

By Jeffrey L Cummings, Serge Gauthier

Given the improvement of latest healing thoughts and items, Serge Gauthier and Jeffrey Cummings have compiled an inventory of latest issues so that it will be of curiosity essentially to neurologists targeting Alzheimer's illness, and in addition to psychiatrists and geriatricians. The members, all revered of their subspecialties, have written updated, comprehensively referenced chapters which should offer information in addition to stimulate dialogue on the place present remedy is heading.

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Extra resources for Alzheimer's disease and related disorders annual, 2002

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40 It has been suggested that neurons undergoing apoptosis might release more β-amyloid (Aβ) leading to an acceleration of the disease process. It is widely accepted that the Aβ peptide is the principal toxic agent in AD. 1,41–46 Surprisingly, studies on cell culture systems in which the cells have been exposed to Aβ point to the involvement of caspases other than those found to be altered in postmortem AD tissue. Comparison of the results from the various cell culture studies is difficult since they utilize a variety of neuronal cell types, a number of culture conditions and different concentrations, forms and aggregation states of the Aβ peptide.

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Neocortical synapses in a presenile dementia. J Neuropathol Exp Neurol 1967; 26: 150–151. 74. Terry RD, Masliah E, Salmon DP et al. Physical basis of cognitive alterations in Alzheimer’s disease: synapse loss is the major correlate of cognitive impairment. Ann Neurol 1991; 30:572–580. 35 75. Mucke L, Masliah E, Yu GQ et al. High-level neuronal expression of abeta 1–42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation. J Neurosci 2000; 20:4050–4058.

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