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Cockcroft DW, Ruffin RE, Dolovitch J, Hargreave FE. Allergen-induced increase in nonallergic bronchial reactivity. Clin Allergy 1977; 7:503–13. Polosa R, Rorke S, Holgate ST. Evolving concepts on the value of adenosine hyperresponsiveness in asthma and chronic obstructive pulmonary disease. Thorax 2002; 57: 649–54. Cushley MJ, Tattersfield AE, Holgate ST. Inhaled adenosine and guanosine on airway resistance in normal and asthmatic subjects. Br J Clinical Pharmacol 1983; 15: 161–5. 7 Pathology of asthma Introduction The appreciation that even mild asthma was associated with airway inflammation has led to the central recognition that asthma is a chronic inflammatory disorder of the airways.

Comment Although airway inflammation is central to our understanding of asthma, the diagnosis and assessment of asthma on clinical grounds are made with reference to symptoms and lung function rather than measures of airway inflammation. Reliable, reproducible and safe methods to assess the extent of airway inflammation are clearly needed. The technique of sputum induction has been utilized as a research tool for some time and an increasing body of evidence supports its potential utility. However, it is usually performed by research centres with established expertise in the technique.

97), but this did not attain statistical significance. 0). Neither the presence of a family history of asthma, nor atopy was associated with persistent wheeze. 60). 1, Fig. 1). 1 Prognosis and development of recent wheeze, airway hyper-responsiveness (AHR), and atopy in the study sample (n=498) Source: Xuan et al. (2002). Fig. 1 Risk factors for late onset and persistence of recent wheeze. Source: Xuan et al. (2002). Incidence and remission of asthma: a retrospective study on the natural history of asthma.

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